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In particular, a growing body of in vivo evidence supports the concept that some cancers become functionally dependent on FSP1, creating a therapeutic vulnerability that can be exploited either as monotherapy in specific contexts or as combination therapy with ferroptosis-inducing treatments

This difference in subcellular localization, combined with its distinct expression patterns, suggests that the two proteins play different roles in regulating cellular GSH levels (Ward et al., 2024)

If you traveled or experienced increased stress during the holidays, your gut microbiome might need a tune-up for the New Year

Long COVID and MCAS can cause severe food sensitivities due to gut inflammation and mast cell activation